Some people have freckles, and some people do not have freckles. if a child has freckles, at least one parent has freckles. however, the child may have a sibling that does not have freckles. which statement best describes the presence of freckles? freckles are not determined by dna. there are two alleles for freckles. there is no dominant allele for freckles. freckles are determined by two genes instead of by one.

The correct answer is that there are two alleles for freckles.  

On the basis of the occurrence of the freckles, it can be suggested that the freckle refers to an autosomal dominant condition. The freckle can only be inherited if there is a presence of at least one dominant allele in any of the parent.  

In case, if both the parents are heterozygous for the trait, they can generate an offspring devoid of freckle by inheriting the recessive alleles to the progeny. Thus, the freckles are monitored by two alleles, that is, it can be either recessive or dominant.  

Option). there are two alleles for freckles.

Explanation:

Freckles represent clusters of cells with concentrated melanin, generally visible for individuals with fair complexion. Freckles can be transferred from one generation to another as both genetic and environmental factors are associated with freckles.

There are two alleles for freckles, one dominant and one recessive. Freckles represent a dominant trait, which means people with a single dominant allele will have freckles, while people with both recessive alleles will do not have freckles.

So, the correct answer is option). there are tow alleles for freckles.

the father in the first generation probably had the recessive colorblind trait in his x chromosome. if he and his wife both had the colorblind trait, even if she had 1 recessive and 1 dominant, then that trait would be passed on to their kids.

(since you didn't attach the family tree this is just an assumption)

after a the power stroke, adp is released, but the cross-bridge formed is still in place. atp then binds to myosin, moving the myosin to its high-energy state, releasing the myosin head from the actin active site.