You are testing learning and memory in two strains (A and B) of hamsters by determining which hamsters learn and remember how to navigate a maze better. You discover that a strain of hamsters raised in a diverse environment has enhanced memory compared to a strain of hamsters raised in a bare cage. You look at a specific gene that is involved in brain development and is activated during learning and memory. If you use bisulfite treatment and subsequent sequencing to study the methylation pattern is of the promoter for this gene, you find that at the same region of the promoter sequence: Strain A has the following bisulfite treated sequence: GTACGTTAAACGATCG
Strain A has the following untreated sequence: GTACGTTAAACGATCG
Strain B has the following bisulfite treated sequence: GTATGTTAAATGATTG
Strain B has the following untreated sequence: GTACGTTAAACGATCG
Based on the sequences above, greater levels of transcription for this learning and memory gene more likely occur in which of the strains?

In 1959, doctors began using the powerful antibiotic methicillin to treat infections of staphylococcus aureus, but within two years, methicillin-resistant strains of s. aureus (mrsa) appeared. how did the resistant strains of s. aureus emerge? in 1959, doctors began using the powerful antibiotic methicillin to treat infections of staphylococcus aureus, but within two years, methicillin-resistant strains of s. aureus (mrsa) appeared. how did the resistant strains of s. aureus emerge? staphylococcus aureus bacteria that were able to synthesize cell walls using a protein that was not affected by methicillin survived the methicillin treatments and reproduced at higher rates than did other individuals. over time, these resistant individuals became increasingly common. in response to treatment of staphylococcus aureus infections with methicillin, some bacteria began to synthesize cell walls using a protein that was not affected by methicillin. these bacteria survived the methicillin treatments and reproduced at higher rates than did other individuals. over time, these resistant individuals became increasingly common. in response to treatment of staphylococcus aureus infections with methicillin, bacterial populations gradually began to synthesize cell walls using a protein that was not affected by methicillin.