In a series of experiments, genes that code for mutant forms of a receptor tyrosine kinase (rtk) are introduced into cells. the cells also can express their own normal form of the receptor from their normal gene, but the mutant genes are constructed so that the mutant rtk is expressed at considerably higher concentration than the normal rtk. what would be the consequences of introducing a mutant gene that codes for an rtk

a) lacking its extracellular domain?

b) lacking its intracellular domain?